Publications and pre-prints

Below are some of our selected publications and pre-prints, for a full list please click here.


Diverse lateral gating mechanisms in human rhomboids

In collaboration with the Corey lab, we find that human rhomboids have unexpected diversity in substrate engagement mechanisms. This is most notable with the orphans RHBDL1 and RHBDL3, which have unusually narrow lateral gates that require substantially higher energy to open (currently available as a pre-print on bioRxiv).

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TMD plasticity controls PINK1 import and cleavage

In collaboration with the Collinson and Corey labs in Bristol, we discovered that structural plasticity in the TMD of PINK1, a kinase mutated in Parkinson’s disease, underpins whether it is cleaved by the rhomboid PARL, or enters the matrix of mitochondria.

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RHBDL2 and Orai1 

We performed a bioinformatics-led cell biological screen to identify new substrates for the rhomboid protease RHBDL2. It led to the discovery that RHBDL2 recognises a calcium channel called Orai1 when it is inappropriately activated, and cleaves it to turn it off and thereby control downstream signalling. 

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iRhom2 and ADAM17

We dissected the molecular mechanisms underlying the release of important signalling molecules such as TNF and EGF-like ligands. We revealed that phosphorylation of iRhom2 controls the cell surface sheddase activity of the protease TACE/ADAM17.

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E-cadherin cleavage and tumorigenesis

We noted that during oncogene-driven single cell extrusion from a wild type epithelial monolayer, the adherens junction protein E-cadherin was cleaved. This led us to generate an artificially cleavable form of E-cadherin and show that its cleavage is sufficient to drive epithelial extrusion.

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Secretase rhomboid proteases

We wrote a review of what is (was!) known about rhomboid intramembrane proteases in the mammalian secretory pathway – highlighting their known substrates and forms of regulation.  

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